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Why Immunization Against Covid-19 Causes Heart Attack?

The so called protective function of the vaccine against Covid-19 is based on the fact that the Spyke protein of Covid-19 coming into contact with our immune system causes the production of antibodies which react with this spyke protein.

The human macrophage incorporates this spyke protein on its cell membrane and presents it to the B-cells which produce antibodies against it.

Of course we should not forget that this spyke protein has a structure which responds to the ACE-2 receptor. This means that those Macrophages could connect themselves directly with the corresponding ACE-2 receptor through the spyke protein which is expressed on their cell membrane, causing a quick non-specific immune reaction directly against vascular endothelial cells in the heart and kidneys, but also in respiratory epithelia and in the gastrointestinal tract. Perhaps this is the cause of the very quick and sharpened reaction of many people to the vaccine against Covid-19 which is often connected with later heart disease.

In our practice as general practitioners we often speak to people which connect the first symptoms of myocardial infarction with the application of the Covid-19 vaccine of any type.

Connecting macrophages with vascular endothelial cells causes an inflammation of the coronary vessels, resulting as a vasculitis or myocardial infarction

Of course we have the same result also if we present the Covid-19 directly as an infection with the virus, because its spyke protein is also an important part of the inducing of the immune response to its existence.

The spyke protein expressed on the macrophage cell membrane causes the production of antibodies by the B-cells. Those antibodies connect with it, but we should not forget that they could connect to ACE-inhibitors, which are used in the therapy of hypertension, thus lowering their effects. Perhaps this is one of the reasons why heart patients with ACE-inhibitors in their therapy tend to react worse to Covid-19 infection, but also do not profit a lot by the vaccine.

The result of the vaccine against Covid-19, or the infection cause by it induces the production of antibodies which react against ACE-inhibitors thus neutralizing their effect.

Of course this is only the molecular point of view.

The Implication of antibodies against the ACE-2 receptor which block the connection of Covid-19 with it may also be dangerous, because those foreign antibodies could be incorporated in the macrophage membrane, causing clustering of macrophages which have the spike protein on their cell membrane with macrophages which contain the corresponding structure of the foreign antibodies. Again is induced a severe autoimmune inflammation of the coronary vessel.

Antibodies which connect with the foreign antibodies against ACE2 Receptors also reduce the function of ACE-inhibitors causing often the increase of arterial hypertonia.


https://www.amazon.com/Immunization-Against-Covid-19-Causes-Autoimmunity-ebook/dp/B0C57FTC7Z/ref=sr_1_2?crid=1WNZ8IOY2PJRU&dib=eyJ2IjoiMSJ9.5wrdOsjvqcIlR1eWM7BwvpKGZScR--CupFVOtDU1VrYkt6LDQvP2GhtI5xSDACqvIKmE-4SiRnM9x2-RGhMXXVgaoIpZHBLPqVwnJzIGWP8.l0Lk74hevPc9aifuYIeS35Ayksizu0BCNtk7js9sRlg&dib_tag=se&keywords=tanya+guleria+theory+of+autoimmunity&qid=1730545376&sprefix=%2Caps%2C186&sr=8-2

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