The so called protective function of the vaccine against Covid-19 is based on the fact that the Spyke protein of Covid-19 coming into contact with our immune system causes the production of antibodies which react with this spyke protein.
The human macrophage incorporates this spyke
protein on its cell membrane and presents it to the B-cells which produce
antibodies against it.
Of course we should not forget that this
spyke protein has a structure which responds to the ACE-2 receptor. This means
that those Macrophages could connect themselves directly with the corresponding
ACE-2 receptor through the spyke protein which is expressed on their cell
membrane, causing a quick non-specific immune reaction directly against vascular
endothelial cells in the heart and kidneys, but also in respiratory epithelia
and in the gastrointestinal tract. Perhaps this is the cause of the very quick
and sharpened reaction of many people to the vaccine against Covid-19 which is
often connected with later heart disease.
In
our practice as general practitioners we often speak to people which connect
the first symptoms of myocardial infarction with the application of the
Covid-19 vaccine of any type.
Connecting
macrophages with vascular endothelial cells causes an inflammation of the coronary
vessels, resulting as a vasculitis or myocardial infarction
Of
course we have the same result also if we present the Covid-19 directly as an
infection with the virus, because its spyke protein is also an important part
of the inducing of the immune response to its existence.
The
spyke protein expressed on the macrophage cell membrane causes the production
of antibodies by the B-cells. Those antibodies connect with it, but we should
not forget that they could connect to ACE-inhibitors, which are used in the
therapy of hypertension, thus lowering their effects. Perhaps this is one of
the reasons why heart patients with ACE-inhibitors in their therapy tend to
react worse to Covid-19 infection, but also do not profit a lot by the vaccine.
The
result of the vaccine against Covid-19, or the infection cause by it induces
the production of antibodies which react against ACE-inhibitors thus
neutralizing their effect.
Of
course this is only the molecular point of view.
The
Implication of antibodies against the ACE-2 receptor which block the connection
of Covid-19 with it may also be dangerous, because those foreign antibodies
could be incorporated in the macrophage membrane, causing clustering of
macrophages which have the spike protein on their cell membrane with
macrophages which contain the corresponding structure of the foreign antibodies.
Again is induced a severe autoimmune inflammation of the coronary vessel.
Antibodies
which connect with the foreign antibodies against ACE2 Receptors also reduce
the function of ACE-inhibitors causing often the increase of arterial
hypertonia.
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