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Why Ozempic Causes Blindness?

What Is Ozempic? Ozempic (semaglutide) Injection is a  glucagon -like  peptide  1 (GLP-1) receptor  agonist  indicated as an adjunct to diet and exercise to improve glycemic control in adults with type 2  diabetes mellitus . What Are Side Effects of Ozempic? Ozempic may cause serious side effects including: a lump in the neck, difficulty swallowing, cough, shortness of breath, difficulty breathing, upper abdominal pain, nausea, vomiting, blurred vision, spots or dark strings floating in your vision, fluctuating vision, vision loss, dark or empty areas in your vision, shakiness, nervousness, anxiety, sweating, chills, clamminess, irritability, impatience, confusion, fast heartrate, lightheadedness , dizziness, hunger, decreased urination, swelling in your legs, ankles, or feet, fatigue, rash, itching, and shock Get medical help right away, if you have any of the symptoms listed above. Common side effects of Oze
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Therapy of Pneumothorax

 The most common reason for making a thorax drainage is pneumothorax. As a result of severe thorax traumata often the inner pleura layer is dammaged by a fractured rib and this leads to pneumothorax, or air enters between the two layers of the pleura. This is a life threatening situation, because air in the pleural sack leads to compresion of the injured lung, or lungs and the heart itself.  First aid for pneumothorax is the thorax drainage.There are two most common types of thorax drainages: - drainage of Bülau,  The "Bülau-Drain" is based on the Bülau principle and creates a permanent passive suction within a closed system that is based on the Heber-Drain principle. The  pulmonologist  Gotthard Bülau (1835-1900) used this system in 1875 for the first time for the treatment of  pleural empyema . The Bülau-drain has one significant advantage: it is relatively simple in non-clinical conditions, but it is not very effective, because of the entering point of the drain. Usually i
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In Search of The Perfect Sleep Aid

 For many years, modern medicine has been searching for the perfect sleeping pills. Sleeping pills are a special topic in medicine because they have a lot to do with addiction medicine. Of course, not every sleeping pill can cause addiction, but lighter sleeping pills such as valerian tablets, doxylamine succinate (contained in Schlafsterne), melatonin (or Circadin) have short and insufficient effects. Benzodiazepines, on the other hand, relieve anxiety, which often leads to sleep disorders and are often used by GPs as sleeping pills, although this is not correct. Antidepressants such as mirtazapine are more often used as sleeping pills, but patients often complain of fatigue and drowsiness after their use, which is incompatible with driving, for example. Z-drugs are the 3rd choice of sleep medicine, they have a long and sufficient effect, but can be very addictive. There are patients who struggle for years with addiction to zolpidem, or zopiclone. Many GPs give up in this fight and pr
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Auf der Suche der perfekte Schlafmittel

Seit vielen Jahren ist die moderne Medizin auf der Suche der perfekte Schlafmittel. Schlafmittel sind ein besonderes Thema in Medizin, denn sie haben viel zu tun mit Suchtmedizin. Natürlich nicht jedes Schlafmittel  kann Sucht verursachen, aber leichtere Schlafmittel wie z.B. Baldriantabletten,  Doxylamin succinat (beinhaltet in Schlafsterne), Melatonin (oder Circadin) sind mit kurze und unzureichende Wirkung. Benzodiazepine dagegen lösen Ängste, die öfters zu Schlafstörungen führen und sind von Hausärzte öfters als Schlafmittel angewendet obwohl das nicht richtig ist. Öfters als Schlafmittel werden Antidepressanten wie Mirtazapin angewendet, aber die Patienten beklagen öfters nach Ihren Gebrauch Ermüdung und Eingenommenheit, die z.B. nicht mit Autofahren vereinbar sind. Z-Medikamenten sind 3-te Wahl der Schlafmedizin, sie haben lange und zureichende Wirkung, können aber sehr süchtig machen. Es gibt Patienten die jahrelang mit der Sucht nach Zolpidem, oder Zopiclon kämpfen. Viele Hausä
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Why Immunization Against Covid-19 Causes Heart Attack?

The so called protective function of the vaccine against Covid-19 is based on the fact that the Spyke protein of Covid-19 coming into contact with our immune system causes the production of antibodies which react with this spyke protein. The human macrophage incorporates this spyke protein on its cell membrane and presents it to the B-cells which produce antibodies against it. Of course we should not forget that this spyke protein has a structure which responds to the ACE-2 receptor. This means that those Macrophages could connect themselves directly with the corresponding ACE-2 receptor through the spyke protein which is expressed on their cell membrane, causing a quick non-specific immune reaction directly against vascular endothelial cells in the heart and kidneys, but also in respiratory epithelia and in the gastrointestinal tract. Perhaps this is the cause of the very quick and sharpened reaction of many people to the vaccine against Covid-19 which is often connected with late
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CoViD-19 and ACE inhibitors

  The disease is often moderate or even asymptomatic. However, severe courses can occur, usually manifesting as pneumonia. In some of the seriously ill patients, severe cardiovascular damage is also observed. For people with heart disease, the disease - which is caused by the SARS-CoV-2 virus - thus appears to be particularly dangerous. Angiotensin-converting enzyme 2 (ACE2) plays a major role in the body's water balance. ACE inhibitors lower blood pressure and reduce afterload - which also makes them very useful in treating heart failure disease. ACE2 are particularly abundant in the heart and lungs. Currently, attention is focused on this enzyme because ACE2 has been identified as a functional receptor for the coronaviruses SARS-CoV and SARS-CoV-2. It appears that the amount of ACE2 increases by taking ACE2 inhibitors in response. Thus, the body responds to inhibition of these receptors by increasing potential docking sites. It has not yet been proven whether this mec
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Angiotensin converting enzyme 2

Angiotensin converting enzyme 2, or ACE2, is an exopeptidase expressed primarily by vascular endothelial cells in the heart and kidneys, but also in respiratory epithelia[1] and in the gastrointestinal tract. It is the target of several coronaviruses, including SARS-CoV and SARS-CoV-2. 2 Biochemistry ACE2 is a transmembrane metallocarboxypeptidase composed of 805 amino acids. Zinc and chloride ions act as cofactors. The extracellular region consists of two domains, a zinc metallopeptidase domain and a C-terminal collectrin homology domain. The enzyme exhibits homology to angiotensin converting enzyme (ACE). ACE2 is encoded by the ACE2 gene on the X chromosome (gene locus Xp22.2). In addition to being expressed as a transmembrane protein, a soluble form exists in serum. 3 Function ACE2 cleaves angiotensin II into angiotensin (1-7), which has anti-inflammatory and lung protective effects via MAS and AT2 receptors. 4 Clinical 4.1 Infectiology ACE2 serves as a major entry p
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